Acute Kidney Injury (AKI)

Nephrology

Overview

Definition

A sudden decrease in renal function, resulting in the retention of urea and other nitrogenous waste products and in the dysregulation of extracellular volume and electrolytes.

Epidemiology

Occurs in ~5-7% of all hospitalized patients and up to 50% of ICU patients.

Etiology & Risk Factors

  • Prerenal (hypovolemia, heart failure, sepsis)
  • Intrinsic (Acute Tubular Necrosis, Acute Interstitial Nephritis, Glomerulonephritis)
  • Postrenal (BPH, kidney stones, foley catheter obstruction)

Clinical Symptoms

  • Oliguria or anuria
  • Edema
  • Fatigue
  • Confusion (uremia)
  • Often asymptomatic in early stages

Clinical Approach

Diagnosis

  • Increase in serum creatinine by ≥ 0.3 mg/dL within 48 hrs
  • Increase in serum Cr to ≥ 1.5 times baseline
  • Urine volume < 0.5 mL/kg/h for 6 hours
  • FENa (Fractional Excretion of Sodium) to differentiate pre-renal from intrinsic
  • Renal ultrasound (rule out obstruction)

Management

  • Treat the underlying cause
  • Optimize volume status (IV fluids if pre-renal)
  • Avoid nephrotoxins (NSAIDs, contrast, aminoglycosides)
  • Renal Replacement Therapy (Dialysis) if indicated (AEIOU criteria)

Complications

  • Hyperkalemia
  • Metabolic acidosis
  • Volume overload / Pulmonary edema
  • Uremic encephalopathy
  • Progression to Chronic Kidney Disease