Rapidly Progressive Glomerulonephritis (RPGN)

Nephrology / Critical Care

Overview

Definition

A severe nephritic syndrome characterized by rapid, progressive decline in renal function over weeks to months, histologically characterized by crescent formation in most glomeruli.

Epidemiology

Rare but life-threatening emergency. Classified into three main immunopathologic types.

Etiology & Risk Factors

  • Type 1: Anti-GBM disease (Goodpasture Syndrome)
  • Type 2: Immune-complex mediated (Lupus, IgA, PSGN)
  • Type 3: Pauci-immune (ANCA-associated vasculitis: GPA, MPA, EGPA)

Clinical Symptoms

  • Rapid-onset oliguria or anuria
  • Gross hematuria
  • Uremic symptoms (nausea, confusion, pericarditis)
  • Hemoptysis (in Goodpasture syndrome or ANCA-associated vasculitis)

Clinical Approach

Diagnosis

  • Serology: Anti-GBM antibodies, ANCA, ANA, complements
  • Renal biopsy showing cellular crescents in Bowman's space on LM, and linear IgG (Type 1), granular (Type 2), or negative (Type 3) immunofluorescence

Management

  • Pulse intravenous corticosteroids (Methylprednisolone)
  • Cyclophosphamide
  • Plasmapheresis (critical in Anti-GBM disease to remove circulating antibodies)

Complications

  • End-stage renal disease (often requiring immediate dialysis)
  • Pulmonary hemorrhage (fatal if untreated)
  • Severe uremia